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Its research profile centers on mitochondrial retrograde signaling, where the peptide is thought to help regulate how cells adjust energy production in response to metabolic stress, independent of any hormone-receptor cascade

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The diagnosis of glutathione synthetase deficiency is established by: Low activity of glutathione synthetase in cultured skin fibroblasts and/or red blood cells

Kurtzman, N

MOTS-c functions as a mitochondrial signaling peptide. It travels from the mitochondria to the rest of the cell and even to other parts of the body, influencing how cells use energy