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The reason retatrutide adds GCGR activity is that glucagon receptor activation in the liver also drives fatty acid oxidation and, when combined with GLP-1 and GIP agonism, produces a metabolic phenotype that single- and dual-receptor agonism do not

doi: 10.1016/j.hsr.2025.100237 88

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Peer review Peer review information Nature Chemistry thanks Daniel Reker and the other, anonymous, reviewer(s) for their contribution to the peer review of this work

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