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This evidence suggested that high glucose induces the activation of PSCs and promotes their proliferation, leading to increased ECM secretion

In contrast, GIP receptors (GIPR) were localized only in the circular and longitudinal muscular layers of jejunum

Several point mutations (V1016G, V1016I and F1269C) were detected [178, 180]

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The intricate underlying etiology involves hyperglycemia, hemoglobin glycation, impaired neutrophil function, dysregulated macrophage polarization and function, sustained production of proinflammatory cytokines, impaired angiogenesis, reduced fibroblast and keratinocyte proliferation and migration, compromised growth factor production, decreased cytokine production, downregulated MMPs production, neuropathy, and nitrous oxide blockade [133, 158, 160]