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Furthermore, EA attenuated pathological damage and neuronal apoptosis in rats with TBI, leading to gradual improvement in pathological morphology, such as nuclear consolidation, fragmentation, cell swelling, neuronal necrosis, and scar tissue

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doi: 10.1053/j.ajkd.2020.05.015 172

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Degradation of glucagon-like peptide-1 by human plasma in vitro yields an N-terminally truncated peptide that is a major endogenous metabolite in vivo