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As such, DATs selectively protect hepatocytes while inducing HSC cuproptosis, thereby exerting antifibrotic effects [235]

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Although bile-based TCMs are multi-component and exert milder effects than synthetic agonists, their core ligands, such as TCA and GCA, have the potential to trigger similar hepatotoxic responses if they accumulate excessively and over-activate intrahepatic FXR signaling